Karolinska Institute-ko ikertzaileekin lankidetzan egindako argitalpenak (37)

2024

  1. Signal peptide peptidase-like 2b modulates the amyloidogenic pathway and exhibits an Aβ-dependent expression in Alzheimer's disease

    Progress in Neurobiology, Vol. 235

2023

  1. ARG1-expressing microglia show a distinct molecular signature and modulate postnatal development and function of the mouse brain

    Nature Neuroscience, Vol. 26, Núm. 6, pp. 1008-1020

  2. Targeting galectin-3 to counteract spike-phase uncoupling of fast-spiking interneurons to gamma oscillations in Alzheimer’s disease

    Translational Neurodegeneration, Vol. 12, Núm. 1

  3. Timing to be precise? An overview of spike timing-dependent plasticity, brain rhythmicity, and glial cells interplay within neuronal circuits

    Molecular Psychiatry, Vol. 28, Núm. 6, pp. 2177-2188

2022

  1. Abilities of the BRICHOS domain to prevent neurotoxicity and fibril formation are dependent on a highly conserved Asp residue

    RSC Chemical Biology

  2. S100A9 amyloid growth and S100A9 fibril-induced impairment of gamma oscillations in area CA3 of mouse hippocampus ex vivo is prevented by Bri2 BRICHOS

    Progress in Neurobiology, Vol. 219

2021

  1. Ablation of p75NTR signaling strengthens gamma–theta rhythm interaction and counteracts Aβ-induced degradation of neuronal dynamics in mouse hippocampus in vitro

    Translational Psychiatry, Vol. 11, Núm. 1

  2. Amphetamine sensitization alters hippocampal neuronal morphology and memory and learning behaviors

    Molecular Psychiatry, Vol. 26, Núm. 9, pp. 4784-4794

  3. Bri2 BRICHOS chaperone rescues impaired fast-spiking interneuron behavior and neuronal network dynamics in an AD mouse model in vitro

    Neurobiology of Disease, Vol. 159

  4. Guidelines for the use and interpretation of assays for monitoring autophagy (4th edition)1

    Autophagy, Vol. 17, Núm. 1, pp. 1-382

  5. Impaired spike-gamma coupling of area CA3 fast-spiking interneurons as the earliest functional impairment in the App NL-G-F mouse model of Alzheimer’s disease

    Molecular Psychiatry, Vol. 26, Núm. 10, pp. 5557-5567

  6. Selective deletion of Caspase-3 gene in the dopaminergic system exhibits autistic-like behaviour

    Progress in Neuro-Psychopharmacology and Biological Psychiatry, Vol. 104

2020

  1. Augmentation of Bri2 molecular chaperone activity against amyloid-β reduces neurotoxicity in mouse hippocampus in vitro

    Communications Biology, Vol. 3, Núm. 1

  2. Capsaicin-Induced Impairment of Functional Network Dynamics in Mouse Hippocampus via a TrpV1 Receptor-Independent Pathway: Putative Involvement of Na+/K+-ATPase

    Molecular Neurobiology, Vol. 57, Núm. 2, pp. 1170-1185

  3. Copy number variants (CNVs): a powerful tool for iPSC-based modelling of ASD

    Molecular autism, Vol. 11, Núm. 1, pp. 42

  4. Existence of FGFR1-5-HT1AR heteroreceptor complexes in hippocampal astrocytes. Putative link to 5-HT and FGF2 modulation of hippocampal gamma oscillations

    Neuropharmacology, Vol. 170

  5. Modulation of Kv3.1/Kv3.2 promotes gamma oscillations by rescuing Aβ-induced desynchronization of fast-spiking interneuron firing in an AD mouse model in vitro

    Journal of Physiology, Vol. 598, Núm. 17, pp. 3711-3725

2019

  1. Oestrogen receptor β mediates the actions of bisphenol-A on ion channel expression in mouse pancreatic beta cells

    Diabetologia, Vol. 62, Núm. 9, pp. 1667-1680

2018

  1. Receptor–receptor interactions in multiple 5-HT1A heteroreceptor complexes in raphe-hippocampal 5-HT transmission and their relevance for depression and its treatment

    Molecules, Vol. 23, Núm. 6

  2. TrpV1 receptor activation rescues neuronal function and network gamma oscillations from Aβ-induced impairment in mouse hippocampus in vitro

    eLife, Vol. 7