Publicaciones (41) Publicaciones de Mª José Sánchez Sanz

2023

  1. Expresión de factor V de la coagulación en distintas etapas del desarrollo embrionario en ratón

    LXIV Congreso Nacional de la SEHH. XXXVIII Congreso Nacional SETH. 38th World Congress of the International Society of Hematology (ISTH)

  2. Therapeutic potential of fetal liver cell transplantation in hemophilia A mice

    Haematologica, Vol. 108, Núm. 6, pp. 1544-1554

2022

  1. Single versus tandem autologous stem-cell transplantation in patients with newly diagnosed multiple myeloma and high-risk cytogenetics. A retrospective, open-label study of the PETHEMA/Spanish Myeloma Group (GEM)

    Leukemia and Lymphoma, Vol. 63, Núm. 14, pp. 3438-3447

  2. The Vascular Endothelium and Coagulation: Homeostasis, Disease, and Treatment, with a Focus on the Von Willebrand Factor and Factors VIII and V

    International journal of molecular sciences, Vol. 23, Núm. 15

2018

  1. Searching for a Cell-Based Therapeutic Tool for Haemophilia A within the Embryonic/Foetal Liver and the Aorta-Gonads-Mesonephros Region

    Thrombosis and Haemostasis, Vol. 118, Núm. 8, pp. 1370-1381

2017

  1. A population of hematopoietic stem cells derives from GATA4-expressing progenitors located in the placenta and lateral mesoderm of mice

    Haematologica, Vol. 102, Núm. 4, pp. 647-655

  2. Characterization of a Fetal Liver Cell Population Endowed with Long-Term Multiorgan Endothelial Reconstitution Potential

    Stem Cells, Vol. 35, Núm. 2, pp. 507-521

2013

  1. Single site-specific integration targeting coupled with embryonic stem cell differentiation provides a high-throughput alternative to in vivo enhancer analyses

    Biology Open, Vol. 2, Núm. 11, pp. 1229-1238

2010

  1. Cis-regulatory remodeling of the SCL locus during vertebrate evolution

    Molecular and Cellular Biology, Vol. 30, Núm. 24, pp. 5741-5751

  2. Enhanced hematovascular contribution of SCL 3′ enhancer expressing fetal liver cells uncovers their potential to integrate in extramedullary adult niches

    Stem Cells, Vol. 28, Núm. 1, pp. 100-112

2009

  1. Cell fusion contributes to pericyte formation after stroke

    Journal of Cerebral Blood Flow and Metabolism, Vol. 29, Núm. 3, pp. 480-485

  2. In vivo fate mapping with SCL regulatory elements identifies progenitors for primitive and definitive hematopoiesis in mice

    Mechanisms of Development, Vol. 126, Núm. 10, pp. 863-872

2008

  1. A novel mode of enhancer evolution: The Tal1 stem cell enhancer recruited a MIR element to specifically boost its activity

    Genome Research, Vol. 18, Núm. 9, pp. 1422-1432

  2. Organ-injury-induced reactivation of hemangioblastic precursor cells

    Leukemia, Vol. 22, Núm. 1, pp. 103-113

2007

  1. The notch pathway positively regulates programmed cell death during erythroid differentiation

    Leukemia, Vol. 21, Núm. 7, pp. 1496-1503

2006

  1. The RNA binding protein Zfp36l1 is required for normal vascularisation and post-transcriptionally regulates VEGF expression

    Developmental Dynamics, Vol. 235, Núm. 11, pp. 3144-3155

2005

  1. Transgenic analysis of the stem cell leukemia +19 stem cell enhancer in adult and embryonic hematopoietic and endothelial cells

    Stem Cells, Vol. 23, Núm. 9, pp. 1378-1388

2004

  1. Kidney, blood, and endothelium: Developmental expression of stem cell leukemia during nephrogenesis

    Kidney International, Vol. 65, Núm. 4, pp. 1162-1169

  2. The scl +18/19 Stem Cell Enhancer Is Not Required for Hematopoiesis: Identification of a 5′ Bifunctional Hematopoietic-Endothelial Enhancer Bound by Fli-1 and Elf-1

    Molecular and Cellular Biology, Vol. 24, Núm. 5, pp. 1870-1883

2003

  1. Manipulation of Mouse Hematopoietic Progenitors by Specific Retroviral Infection

    Journal of Biological Chemistry, Vol. 278, Núm. 44, pp. 43556-43563