Publicaciones en colaboración con investigadores/as de Centro Andaluz de Biología del Desarrollo (15)

2019

  1. Pathophysiological characterization of MERRF patient-specific induced neurons generated by direct reprogramming

    Biochimica et Biophysica Acta - Molecular Cell Research, Vol. 1866, Núm. 5, pp. 861-881

2018

  1. Intracellular cholesterol accumulation and coenzyme Q10 deficiency in Familial Hypercholesterolemia

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1864, Núm. 12, pp. 3697-3713

  2. Therapy with coenzyme Q10

    Coenzyme Q10: Uses, Health Effects and Role in Disease (Nova Science Publishers, Inc.), pp. 1-63

2017

  1. Two coffins and a funeral: early or late caspase activation determines two types of apoptosis induced by DNA damaging agents

    Apoptosis, Vol. 22, Núm. 3, pp. 421-436

2016

  1. AMPK as a target in rare diseases

    Current Drug Targets, Vol. 17, Núm. 8, pp. 921-931

  2. The connections among autophagy, inflammasome and mitochondria

    Current Drug Targets, Vol. 17, Núm. 16

2015

  1. Emerging roles of apoptotic microtubules during the execution phase of apoptosis

    Cytoskeleton, Vol. 72, Núm. 9, pp. 435-446

  2. Pharmacological chaperones and coenzyme Q10 treatment improves mutant β-glucocerebrosidase activity and mitochondrial function in neuronopathic forms of gaucher disease

    Scientific Reports, Vol. 5

2014

  1. Clinical applications of coenzyme Q10

    Frontiers in Bioscience - Landmark, Vol. 19, Núm. 4, pp. 619-633

  2. PEGylated versus non-PEGylated magnetic nanoparticles as camptothecin delivery system

    Beilstein Journal of Nanotechnology, Vol. 5, Núm. 1

2013

  1. Apoptotic microtubules delimit an active caspase free area in the cellular cortex during the execution phase of apoptosis

    Cell Death and Disease, Vol. 4, Núm. 3

2012

  1. Recovery of MERRF Fibroblasts and Cybrids Pathophysiology by Coenzyme Q10

    Neurotherapeutics, Vol. 9, Núm. 2, pp. 446-463

  2. Screening of effective pharmacological treatments for MELAS syndrome using yeasts, fibroblasts and cybrid models of the disease

    British Journal of Pharmacology, Vol. 167, Núm. 6, pp. 1311-1328

2011

  1. Apoptotic microtubule network organization and maintenance depend on high cellular ATP levels and energized mitochondria

    Apoptosis, Vol. 16, Núm. 4, pp. 404-424

  2. Secondary coenzyme Q 10 deficiency triggers mitochondria degradation by mitophagy in MELAS fibroblasts

    FASEB Journal, Vol. 25, Núm. 8, pp. 2669-2687