Publicaciones en colaboración con investigadores/as de Universidad de Sevilla (13)

2019

  1. Pantothenate Rescues Iron Accumulation in Pantothenate Kinase-Associated Neurodegeneration Depending on the Type of Mutation

    Molecular Neurobiology, Vol. 56, Núm. 5, pp. 3638-3656

2017

  1. Amitriptyline down-regulates coenzyme Q10biosynthesis in lung cancer cells

    European Journal of Pharmacology, Vol. 797, pp. 75-82

  2. Two coffins and a funeral: early or late caspase activation determines two types of apoptosis induced by DNA damaging agents

    Apoptosis, Vol. 22, Núm. 3, pp. 421-436

2016

  1. Amitriptyline induces mitophagy that precedes apoptosis in human HepG2 cells

    Genes and Cancer, Vol. 7, Núm. 7-8, pp. 260-277

  2. Targeting autophagy and mitophagy for mitochondrial diseases treatment

    Expert Opinion on Therapeutic Targets, Vol. 20, Núm. 4, pp. 487-500

2015

  1. Critical role of AMP-activated protein kinase in the balance between mitophagy and mitochondrial biogenesis in MELAS disease

    Biochimica et Biophysica Acta - Molecular Basis of Disease, Vol. 1852, Núm. 11, pp. 2535-2553

  2. Emerging roles of apoptotic microtubules during the execution phase of apoptosis

    Cytoskeleton, Vol. 72, Núm. 9, pp. 435-446

  3. Pharmacological chaperones and coenzyme Q10 treatment improves mutant β-glucocerebrosidase activity and mitochondrial function in neuronopathic forms of gaucher disease

    Scientific Reports, Vol. 5

  4. The Apoptotic Microtubule Network During the Execution Phase of Apoptosis

    CELL DEATH - AUTOPHAGY, APOPTOSIS AND NECROSIS (INTECH EUROPE), pp. 299-329

2014

  1. Apoptotic cells subjected to cold/warming exposure disorganize apoptotic microtubule network and undergo secondary necrosis

    Apoptosis, Vol. 19, Núm. 9, pp. 1364-1377

  2. Clinical applications of coenzyme Q10

    Frontiers in Bioscience - Landmark, Vol. 19, Núm. 4, pp. 619-633

  3. Coenzyme Q10 therapy

    Molecular Syndromology, Vol. 5, Núm. 3-4, pp. 187-197

2013

  1. Apoptotic microtubules delimit an active caspase free area in the cellular cortex during the execution phase of apoptosis

    Cell Death and Disease, Vol. 4, Núm. 3