Publikationen, an denen er mitarbeitet Pedro Bullón (35)

2021

  1. Inhibition of the NLRP3 inflammasome prevents ovarian aging

    Science Advances, Vol. 7, Núm. 1

  2. Potential role of the mitochondria for the dermatological treatment of papillon-lefèvre

    Antioxidants, Vol. 10, Núm. 1, pp. 1-9

2020

  1. A diet rich in saturated fat and cholesterol aggravates the effect of bacterial lipopolysaccharide on alveolar bone loss in a rabbit model of periodontal disease

    Nutrients, Vol. 12, Núm. 5

  2. Blockade of the NLRP3 inflammasome improves metabolic health and lifespan in obese mice

    GeroScience, Vol. 42, Núm. 2, pp. 715-725

  3. Is AMP-Activated Protein Kinase Associated to the Metabolic Changes in Primary Ovarian Insufficiency?

    Antioxidants and Redox Signaling, Vol. 33, Núm. 15, pp. 1115-1121

  4. Molecular inflammation and oxidative stress are shared mechanisms involved in both myocardial infarction and periodontitis

    Journal of Periodontal Research, Vol. 55, Núm. 4, pp. 519-528

  5. NLRP3 Inflammasome Inhibition by MCC950 in Aged Mice Improves Health via Enhanced Autophagy and PPARα Activity

    The journals of gerontology. Series A, Biological sciences and medical sciences, Vol. 75, Núm. 8, pp. 1457-1464

  6. NLRP3 inflammasome suppression improves longevity and prevents cardiac aging in male mice

    Aging Cell, Vol. 19, Núm. 1

2018

  1. Autophagic dysfunction in patients with Papillon-Lefèvre syndrome is restored by recombinant cathepsin C treatment

    Journal of Allergy and Clinical Immunology, Vol. 142, Núm. 4, pp. 1131-1143.e7

  2. Body fat and metabolic age as indicators of inflammation and cardiovascular risk

    European Journal of Preventive Cardiology

  3. Cardiovascular diseases, NLRP3 inflammasome, and western dietary patterns

    Pharmacological Research, Vol. 131, pp. 44-50

2017

  1. Adenosine monophosphate (AMP)-activated protein kinase: A new target for nutraceutical compounds

    International Journal of Molecular Sciences, Vol. 18, Núm. 2

  2. Antidepressants induce autophagy dependent-NLRP3-inflammasome inhibition in Major depressive disorder

    Pharmacological Research, Vol. 121, pp. 114-121

  3. Could NLRP3-Inflammasome Be a Cardiovascular Risk Biomarker in Acute Myocardial Infarction Patients?

    Antioxidants and Redox Signaling, Vol. 27, Núm. 5, pp. 269-275

  4. NLRP3-inflammasome inhibition prevents high fat and high sugar diets-induced heart damage through autophagy induction

    Oncotarget, Vol. 8, Núm. 59, pp. 99740-99756

2016

  1. AMPK Phosphorylation Modulates Pain by Activation of NLRP3 Inflammasome

    Antioxidants and Redox Signaling, Vol. 24, Núm. 3, pp. 157-170

  2. Coenzyme Q Protects Against Age-Related Alveolar Bone Loss Associated to n-6 Polyunsaturated Fatty Acid Rich-Diets by Modulating Mitochondrial Mechanisms

    Journals of Gerontology - Series A Biological Sciences and Medical Sciences, Vol. 71, Núm. 5, pp. 593-600

  3. Gene Expression Profile in Major Depressive Disorder Shows Reduced Mitochondrial Biogenesis

    CNS Neuroscience and Therapeutics

  4. Mutation in cytochrome b gene of mitochondrial DNA in a family with fibromyalgia is associated with NLRP3-inflammasome activation

    Journal of Medical Genetics, Vol. 53, Núm. 2, pp. 113-122

  5. Stress-Induced Depressive Behaviors Require a Functional NLRP3 Inflammasome

    Molecular Neurobiology, Vol. 53, Núm. 7, pp. 4874-4882